The Cardioprotective Effect Of Human Glucagon‐Like Peptide1 Receptor Agonist (Semaglutide) on Cisplatin‐Induced Cardiotoxicity in Rats: Targeting Mitochondrial Functions, Dynamics, Biogenesis, And Redox Status Pathways

Document Type : Abstracts

Abstract

The cardiotoxic effect of chemotherapeutic agents as cisplatin has become a major issue recently. Interference with mitochondrial dynamics, biogenesis, redox status, and apoptosis are the most possible underlying mechanisms.